Why supplement?
Marked asPrimarySupporting
Helps sulfite metabolism
Sulfite oxidase oxidizes sulfite from sulfur-amino-acid metabolism to sulfate so it can be excreted; this enzyme depends on the molybdenum cofactor. Legumes, grains, and nuts already contain enough molybdenum, and dietary absorption is good. Sulfur-amino-acid metabolism produces sulfite every day; molybdenum lets this oxidation path finish.
Supports purine breakdown
Xanthine oxidase turns hypoxanthine through xanthine into uric acid; aldehyde oxidase metabolizes many aldehydes and drug intermediates; both enzymes contain the molybdenum cofactor. Need is extremely low and a mixed diet suffices. If molybdenum appears in a supplement, the function is still to maintain these oxidases so nucleic-acid breakdown and aldehyde detoxification can finish.
How to take it?
Self-starting high doses is not recommended for general wellness.
How oftenA usual diet already supplies enough molybdenum; follow the product label.
What to watch
Cautions
Molybdenum deficiency is rare; kidney disease: avoid self high doses.
High doses may affect copper metabolism.
Interactions
- Public monographs do not list a clear drug conflict; people on medicines should still ask a clinician first
· High doses may cause stomach discomfort
· High long-term intake may cause gout-like joint problems or copper deficiency.
Long-term dependenceNot addictive; high long-term doses may disrupt copper metabolism.